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{"@_fa":"true","link":[{"@_fa":"true","@ref":"self","@href":"http://api.elsevier.com/content/abstract/scopus_id/84951299285"},{"@_fa":"true","@ref":"author-affiliation","@href":"http://api.elsevier.com/content/abstract/scopus_id/84951299285?field=author,affiliation"},{"@_fa":"true","@ref":"scopus","@href":"http://www.scopus.com/inward/record.url?partnerID=HzOxMe3b\u0026scp=84951299285\u0026origin=inward"},{"@_fa":"true","@ref":"scopus-citedby","@href":"http://www.scopus.com/inward/citedby.url?partnerID=HzOxMe3b\u0026scp=84951299285\u0026origin=inward"},{"@_fa":"true","@ref":"full-text","@href":"http://api.elsevier.com/content/article/eid/1-s2.0-S2221169115002191"}],"prism:url":"http://api.elsevier.com/content/abstract/scopus_id/84951299285","dc:identifier":"SCOPUS_ID:84951299285","eid":"2-s2.0-84951299285","dc:title":"Combined treatment of 3-hydroxypyridine-4-one derivatives and green tea extract to induce hepcidin expression in iron-overloaded \u03b2-thalassemic mice","dc:creator":"Upanan S.","prism:publicationName":"Asian Pacific Journal of Tropical Biomedicine","prism:issn":"22211691","prism:coverDate":"2015-08-03","prism:coverDisplayDate":"August 03, 2015","prism:doi":"10.1016/j.apjtb.2015.09.007","pii":"S2221-1691(15)00219-1","dc:description":"\u00a9 2015 Hainan Medical University. Objective: To evaluate the efficacy of deferiprone (DFP), 1-(N-acetyl-6-aminohexyl)-3-hydroxy-2-methylpyridin-4-one (CM1) or green tea extract (GTE) in enhancing expression of hepatic hepcidin1 (Hamp1) mRNA and relieving iron overload in \u03b2-globin knockout thalassemic mice. Methods: The \u03b2-globin knockout thalassemic mice were fed with a ferrocene-supplemented diet for 2 months and oral administration of deionized water, DFP (50 mg/kg), CM1 (50 mg/kg), GTE (50 mg epigallocatechin 3-gallate equivalent/kg), GTE along with DFP (50 mg/kg), and GTE along with CM1 (50 mg/kg) every day for 3 months. Levels of hepatic. Hamp1 mRNA, plasma non-transferrin bound iron, plasma alanine aminotransferase activity and tissue iron content were determined. Results: All chelation treatments could reduce plasma non-transferrin bound iron concentrations. Additionally, hepatic. Hamp1 mRNA expression was significantly up-regulated in the mice in a GTE + DFP combined treatment, correlating with a decrease in the plasma alanine aminotransferase activity and tissue iron deposition. Conclusions: The GTE + DFP treatment could ameliorate iron overload and liver oxidative damage in non-transfusion dependent \u03b2-thalassemic mice, by chelating toxic iron in plasma and tissues, and increasing hepcidin expression to inhibit duodenal iron absorption and iron release from hepatocytes and macrophages in the spleen. 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